Abstract:
The health of Tsurcana sheep in different pastoral ecosystems is the result of a continuous adaptive metabolic process to macro and microclimate changes, depending on individual factors and breed characteristics (the rustic, indigenous breeds are better adapted). In this paper, the biological study material were two-year old Tsurcana sheep raised in Fagaraș, Rucar, Bacau (Comanești area); exclusively pasture fed; from each region and from each flock we collected blood samples from 5 sheep and we presented the average of the values obtained. We found: hypercholesterolemia in the Tsurcana sheep in all three regions (Fagaras and Rucar with similar values), hyperglobulinemia in Tsurcana sheep from Rucar; increased GOT activity in all the Tsurcana tested, most notably at Rucar; increased GPT activity, the highest value in those from Bacau; the increase in GGT activity, the highest value in Ţurcanele de Bacau. This increased plasma activity is due to hepatic lesions, hyperuraemia (the highest values being registered for the Rucar and Bacău Tsurcana); hypercreatinemia (the highest value in Bacau). A classification, depending on the affected organs: the liver is affected in sheep in Rucar and in Bacau; - the kidney and implicitly the nucleoproteic metabolism is more affected in Bacău and Rucăr sheep; the proteic metabolism in sheep in Rucar, where the highest globulin value were identified; on the other hand the increased globulins play a role in the host immunity and we must not forget that the research was carried out during lactation and the sheep from Rucar graze during summer at Lake Iezer at an altitude of over 1800 m; as for cholesterol, it is increased in sheep in all three regions; so lipid metabolism is disrupted, implicitly liver function. In conclusion: Fagaras Tsurcana have hypercholesterolemia, but excretion and epuration are less affected; correlating the obtained results, it can be argued that routine explorations can sometimes reveal unexpected and isolated transaminase elevations; these increases may be influenced by excess weight, adaptive liver reactions, cardio-circulatory failure etc.; many of these are not clinically investigated.
